@article {Schoen:2018:2637-8329:254,
title = "Papilledema in Brain AVMs: Pathophysiologic Considerations on the Basis of a Case Report",
journal = "Neurographics",
parent_itemid = "infobike://asnr/ng",
publishercode ="asnr",
year = "2018",
volume = "8",
number = "4",
publication date ="2018-08-01T00:00:00",
pages = "254-257",
itemtype = "ARTICLE",
issn = "2637-8329",
eissn = "2637-8329",
url = "https://asnr.publisher.ingentaconnect.com/content/asnr/ng/2018/00000008/00000004/art00003",
doi = "doi:10.3174/ng.1700062",
author = "Schoen, S.N. and Blackham, K.A. and Zumofen, D.W. and Mariani, L.",
abstract = "We reported on a young adult who presented with rapid vision loss and headaches. Fundoscopy showed bilateral papilledema and central venous congestion on the left. Angiography revealed an unruptured AVM. The AVM was preoperatively embolized endovascularly and then surgically removed.
A residual deep nidus was embolized directly after surgery, which led to complete angiographic cure, which was confirmed on a 1-year postoperative angiography. After surgery, the patient recovered completely, and a one year post interventional angiography confirmed no residual nidus or early
venous drainage.",
}